Re: Criticisms of Blackmore's approach

From: Mark M. Mills (mmills@htcomp.net)
Date: Sun Jun 11 2000 - 20:39:54 BST

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    Date: Sun, 11 Jun 2000 14:39:54 -0500
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    From: "Mark M. Mills" <mmills@htcomp.net>
    Subject: Re: Criticisms of Blackmore's approach
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    Kenneth,

    >If a meme is not a ' natural object ', not a signal, not a mental concept,
    >not an arifact...what is then a ' new meme ' !?
    >Did we ever came up with a term for this !?

    A Lynch-meme (neural meme) is certainly a natural object. It could be
    observed as a 'signal' if one had the technology to visualize 'gate charge
    states' at the synapse level. In general, the neural meme is better seen
    as a feature of the signal generator process and thus mental concepts.
    The neural meme is the genotype, behavior, including mental concepts,
    represent the phenotype.

    In general, one would not describe a Lynch meme as an artifact, but since
    they can be created via training, I guess one could make that claim, too.

    Just like genetics, the Lynch-meme vocabulary is an attempt to characterize
    real features of biology. The synapses can be seen operating like logic
    gates and signal generators. Autophosphoralating kinases molecules are
    the fastest known molecular switches to electric current. Cadherin
    molecules provide the binding features required to weave a highly detailed
    pattern of gates.

    Knowing the molecular dynamics operating at the synapse level may not
    'explain' cognition and culture, but it will undoubtedly constrain the
    models we use. For example, there is a lot of concern among US parents
    regarding 'hyperactive children,' or 'Attention Deficit Disorder' (ADD).
    Parents in America tend to use ADD terminology to justify giving their
    children a variety of psychoactive drugs. There is some controversy
    regarding this treatment. Is ADD trained? Is ADD biological? Is therapy
    a better treatment than psychoactive drugs? Is ADD related to watching
    American TV?

    When addressing issues such as 'ADD,' our biological models of cognition
    become important. I recently read a book called 'Change your brain, change
    your life' by Amen. It presents about 50 stories about Dr. Amen relating
    patient behaviors to various hyperactive/underactive parts of the brain.
    The evidence of brain disfunction is supported by brain imagery (SPECT
    scans). This sort of evidence was unavailable 20 years ago. Dr. Amen
    argues that behavior emerges from biology and contrasts his work from the
    more conventional view that brains are 'pure logic' machines and need not
    be studied (Turing proved that all logic machines can produce the same
    answer if given enough time). In one dramatic case, Dr. Amen has to search
    the country for a surgeon willing to perform brain surgery on a patient.
    Most of the surgeons contacted refused to operate, thinking therapy (talk)
    was more appropriate. In the end, Dr. Amen found the surgeon and the
    patient regained normal behaviors.

    As to 'new memes', they are as easy to describe as 'new genes.' Every time
    a biologist promotes a new category of phenotypic expression, he has
    created a new gene. Similarly, every time someone promotes a new category
    of behavior, they have created a new neural meme.

    >If you don' t like (puzzled by) the word ' transmit ' regarding memes, how
    >do you explain the term you use (replicating) concerning new memes !?
    >What is here replicated !?

    In brief, neural patterns are replicated via interpersonal interaction. In
    the Windsor Knot example, Dad talks to son and shows him how to tie the
    knot.. The words and gestures dad uses stimulate the son's brain and
    establish new patterns of memories (physically related to kinases patterns
    and new synapse connections). Replication of the meme can be proven when
    the son ties the knot on his own.

    Some might argue that this example fails because there is little, if any,
    neural isomorphism between father and son relative to the Windsor Knot. I
    am not sure this is the issue you raise, but I'll elaborate a bit since it
    is probably on your mind.

    Neural memetics can use the same 'classes' of isomorphism used in genetics:
    phenotypic isomorphism, marker isomorphism and sequence isomorphism. The
    classic 'blue eye' gene is an example of phenotypic isomorphism. We can
    show genes exist via population studies, but no sequence isomorphism exists
    at the level of 'blue eyes.' Isomorphism (individual A and B have the
    gene) is entirely defined by phenotypic expression. Scientists have been
    identifying this class of gene for over a century. My Windsor Knot meme is
    an example of replication with this level of isomorphism.

    About 40 years ago, we found techniques allowing sequences within the DNA
    strands to be identified. This initiated work defining genetic isomorphism
    at the level of DNA markers. For example, biologists talk about 'cancer
    genes.' These 'genes' are based on some geneticist observing a statistical
    relationship between DNA markers and cancer rates among individuals
    carrying the marker. The isomorphism of the 'gene' is not a 'DNA
    instruction' isomorphism, but more of a fingerprint isomorphism. The
    markers are generally junk sequences passively carried from one generation
    to the next. If one were to compare chunks of DNA exhibiting 'marker'
    isomophism, there would be vast differences between the two strands.

    For memetics, 'marker isomorphism' is being reported in books like Dr.
    Amen's 'Change your brain, change your life'. The markers are observed by
    various brain scans (Amen uses SPECT scans). Dr. Amen can statistically
    correlate these brain markers with behaviors (memetic phenotypes).

    Sequence isomorphism is determined only at the level of organic molecules.
    The human genome project seeks to define all the human 'genes', but the
    general public fails to realize molecular biologists define 'gene' in terms
    of organic molecules. When the human genome project is complete, we will
    have a list of all the molecules available to the human population.
    Basically, each gene produces one unique molecule. At this level,
    geneticists can take specific DNA sequences and link them to specific
    organic molecules. Above the molecular level, we don't know the
    operational path, but we have to rely on 'marker' isomorphism.

    Memetic sequence isomorphism is probably 20 or 30 years in the future, but
    it isn't to hard to predict a general model for where they will be found.
    The memetic analog for organic molecules is probably going to be
    inter-cellular neural signals. I suspect there will be classes of kinases
    patterns producing isomorphic neural signals. Just as one combines
    molecules to build phenotypic structures, one combines inter-neural signals
    to produce behavior.

    At the memetic sequence level, we probably inherit a starter set via
    genetic operations, but they may turn out to be products of
    self-organization. When replicating the Windsor Knot meme, much of the
    molecular activity probably involves signal conversion (sensation to
    electrical impulse), transmission of signal, memorization of signal and
    finally signal generation. I suspect much of this involves various
    replicating fundamental electrochemical units (feedback loops, memory
    circuits, signal generators, etc), but we are only beginning to discover
    these neural features.

    Does this answer your question about replication?

    Mark

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